Acute Kidney Injury (AKI)

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Last updated: June 19, 2026Bookmark

Acute Kidney Injury (AKI) is a syndrome of decreased renal function which is reflected by an increase in serum creatinine or a decrease in urine output that occurs over hours-days.

KDIGO defines AKI as:

Rise in serum creatinine > 26 umol/L within 48 hours

Rise in serum creatinine > 1.5 times baseline within 7 days

Urine output < 0.5 mL/kg/h for > 6 consecutive hours

AKI has numerous causes which are divided into prerenal, intrarenal, and post-renal causes.

Renal insufficiency is synonymous with azotemia: which is an elevation of BUN and creatinine. If symptoms of renal failure are present the diagnosis shifts to uraemia and renal replacement therapy (RRT) is indicated. Treatment depends on the cause.

AKI has a 50-70% mortality in the setting of sepsis and multiple organ dysfunction. Uncomplicated AKI due to hypotension and drugs has a better prognosis.

Causes of AKI

CategoryCauses
Prerenal causesVolume depletion, reduced cardiac output, systemic vasodilation, NSAIDs, and ACE inhibitors
Intrarenal causesAcute tubular necrosis, acute interstitial, renal artery or vein thrombosis, malignant hypertension, vasculitides, and glomerulonephritis
Post-renal causesObstruction of the renal pelvis to urethra

Staging of acute kidney injury

StageSerum creatinineUrine output
Stage I> 26.5 umol/L (0.3 mg/dL) or 1.5 – 1.9 times baseline< 0.5 ml/kg/h for 6 – 12 hours
Stage II2.0 – 2.9 times baseline< 0.5 ml/kg/h for > 12 hours
Stage III> 353.6 umol/L (4.0 mg/dL) or > 3 times baseline or Renal Replacement Therapy (RRT)< 0.3 ml/kg/h for > 24 hours or anuria for > 12 hours

Prerenal vs intrarenal vs postrenal failure

Prerenal failureTubulointerstitialGlomerularPostrenal failure
U NaLowHigh (> 40)Low/variableLow
FE NaLow (1%)High (> 1%)Low/variableLow (< 1%)
U OsmHighLow (<350) or isosthenuriaLowHigh
BUN:CrHigh (> 20:1)~ 10:1~ 10:1High or high-normal
UrinalysisNon-specificBrown casts, epithelial casts (acute tubular nephritis), eosinophila (acute interstitial nephritis)Red cell casts, hematuria, proteinuriaNone
  • Common causes
  • Signs and symptoms
    • Urinary output changes
      • Oliguria
      • Anuria
    • Disturbed fluids, electrolytes, and acid-base balance
    • Hypovolemia
      • Hypotension (late sign)
      • Tachycardia
      • Reduced urine output
      • Reduced capillary refill (late sign)
      • Non-visible JVP
      • Poor skin turgor (late sign)
      • Daily weight loss
    • Fluid overload
      • Hypertension
      • Increased JVP
      • Crackles
      • Peripheral odema
      • Gallop rhythm
  • Baseline investigations
    • Serum Creatinine:
      • To calculate eGFR
      • KDIGO criteria for diagnosis and staging
    • Extended electrolytes: Na+, K+, Cl-. Ca2+, PO4-3, Uric acid
    • Complete blood count, FTS, PT/PTT, Blood Glucose
    • Urinalysis
      • Presence of blood cells, casts, sediments points towards intrarenal failure
      • Urine osmolality (Uosm)
  • Investigations to determine whether AKI is prerenal, intrarenal, or post-renal
    • Symptoms: Hemorrhage, Diarrhea and vomiting, Reduced fluid intake, Sepsis, Use of NSAIDs, ACEi/ARBs, CCF, Liver failure
    • Fractional excretion of sodium (FeNa): percentage of sodium excreted in the kidney that is filtered in the urine (normal is 1%, not as important)
      • FeNa < 1%: Tubules intact
      • FeNa > 2%: Tubules damaged (ATN)
    • BUN:Cr: ration of BUN in the serum to Cr in the serum (Normal is between 10:1 and 20:1)
      • BUN:Cr > 20:1: Prerenal
    • Urine Sodium (UNa): Concentration of sodium in the urine
  • Investigations to determine whether it is Acute or Chronic kidney disease
    • KUB CT/US:
      • Shrunken kidney = CKD
      • Normal kidney = AKI or DM/HIVAN/Amyloidosis/ Polycystic kidney disease/ Hydronephrosis
    • Complete blood count for anemia due to reduced EPO
    • Bone biochemistry for renal osteodystrophy or secondary hyperparathyroidism
  • Investigations to determine the etiology
    • KUB CT/US: to determine post-renal causes
    • Renal biopsy: to determine intrarenal failure if pre-renal and post-renal causes have been ruled out
  • Contraindications to kidney biopsy
    • Coagulopathy (due to uremia)
    • Solitary kidney
    • Small kidney
    • Severe hypertension (> 140/90 mmHg)
    • Hydronephrosis
    • Multiple cysts
    • Renal infection (Pyelonephritis, which can develop an abscess)
  • Treatment
  • Treatment of Hypovolemia
    • Fluid resuscitation with 250 – 500 mL boluses (up to 2L can be given)
      • Large volumes of normal saline may cause hyperchloraemic acidosis
      • Ringer’s lactate or Hartmanns solution is a more ****balanced fluid. It is used with caution if there is hyperkalemia and oliguria/anuria
      • Blood components if there is blood loss
      • Human albumin in hepatorenal syndrome and as second line to crystalloids in septic shock
  • Treatment of hypervolemia (fluid overload)
    • Oxygen supplementation
    • Fluid restriction
    • Diuretics if there is symptomatic fluid overload
      • Diuretics are potentially harmful when they used to treat oliguria without fluid overload
    • Renal replacement therapy in AKI + fluid overload + oliguria/anuria
  • Treatment of acidosis
    • Severe acidosis should be referred for renal/critical care and renal replacement therapy.
    • Medical management of acidosis with sodium bicarbonate is controversial since bicarbonate can generate CO2 which can cause respiratory acidosis if there is inadequate ventilation.
  • Treatment of Hyperkalemia
    • Hyperkalemia is treated if the concentration is > 6.5 mmol/L or any EKG changes are present.
    • Cardioprotection(10 10 10 )
      • IV Calcium Gluconate 10% 10mL over 10 minutes. Provides carioprotection for 30 – 60 minutes.
    • Bind potassium(10 50)
      • IV 50mL D50 and 10 Units Insulin. Monitor q1h for hypoglycemia
      • Nebulized 10 – 20 mg Salbutamol. Avoid if there is tachyarrhythmia. Low dose of 10 mg in ischemic heart disease.
      • IV sodium bicarbonate 1-2meq/kg
    • Excrete potassium
      • Renal replacement tehrapy
      • Furosemide or torsemide
  • Complications

Drugs and AKI

CategoryExamples
Safe to continue in AKIParacetamol, warfarin, statins, junior aspirin, clopidogrel, and beta-blockers
Worsens AKINSAIDs, aminoglycosides, ACE inhibitors, Angiotensin II receptor antagonists, and diuretics
Increased risk of toxicity with AKIMetformin, lithium, and digoxin
Reference Intervals
Biochemistry
ACTHP: <80 ng/L
ALTP: 5–35 U/L
AlbuminP: 35–50 g/L
AldosteroneP: 100–500 pmol/L
Alk. phosphataseP: 30–130 U/L
α-AmylaseP: 0–180 IU/dL
α-FetoproteinS: <10 kU/L
Angiotensin IIP: 5–35 pmol/L
ADHP: 0.9–4.6 pmol/L
ASTP: 5–35 U/L
BicarbonateP: 24–30 mmol/L
BilirubinP: 3–17 μmol/L
BNPP: <50 ng/L
CRPP: <10 mg/L
CalcitoninP: <0.1 mcg/L
Calcium (ionized)P: 1.0–1.25 mmol/L
Calcium (total)P: 2.12–2.60 mmol/L
ChlorideP: 95–105 mmol/L
CholesterolP: <5.0 mmol/L
VLDLP: 0.128–0.645 mmol/L
LDLP: <2.0 mmol/L
HDLP: 0.9–1.93 mmol/L
Cortisol AMP: 450–700 nmol/L
Cortisol MidnightP: 80–280 nmol/L
CK ♂P: 25–195 U/L
CK ♀P: 25–170 U/L
CreatinineP: 70–100 μmol/L
FerritinP: 12–200 mcg/L
FolateS: 2.1 mcg/L
FSHP: 2–8 U/L ♂; >25 menopause
GGT ♂P: 11–51 U/L
GGT ♀P: 7–33 U/L
Glucose (fasting)P: 3.5–5.5 mmol/L
Growth hormoneP: <20 mu/L
HbA1C (DCCT)B: 4–6%
HbA1C (IFCC)B: 20–42 mmol/mol
Iron ♂S: 14–31 μmol/L
Iron ♀S: 11–30 μmol/L
Lactate (venous)P: 0.6–2.4 mmol/L
Lactate (arterial)P: 0.6–1.8 mmol/L
LDHP: 70–250 U/L
LHP: 3–16 U/L
MagnesiumP: 0.75–1.05 mmol/L
OsmolalityP: 278–305 mosmol/kg
PTHP: 0.8–8.5 pmol/L
PotassiumP: 3.5–5.3 mmol/L
Prolactin ♂P: <450 U/L
Prolactin ♀P: <600 U/L
PSAP: 0–4 mcg/mL
Protein (total)P: 60–80 g/L
Red cell folateB: 0.36–1.44 μmol/L
Renin (erect)P: 2.8–4.5 pmol/mL/h
Renin (recumbent)P: 1.1–2.7 pmol/mL/h
SodiumP: 135–145 mmol/L
TBGP: 7–17 mg/L
TSHP: 0.5–4.2 mU/L
T4P: 70–140 nmol/L
Free T4P: 9–22 pmol/L
TIBCS: 54–75 μmol/L
TriglyceridesP: 0.50–2.3 mmol/L
T3P: 1.2–3.0 nmol/L
Troponin TP: <0.1 mcg/L
Urate ♂P: 210–480 μmol/L
Urate ♀P: 150–390 μmol/L
UreaP: 2.5–6.7 mmol/L
Vitamin B12S: 0.13–0.68 nmol/L
Vitamin DS: 50 nmol/L
Arterial Blood Gases
pH7.35–7.45
PaCO₂4.7–6.0 kPa
PaO₂>10.6 kPa
Base excess±2 mmol/L
Urine
Cortisol (free)<280 nmol/24h
Hydroxyindole acetic acid16–73 μmol/24h
Hydroxymethylmandelic acid16–48 μmol/24h
Metanephrines0.03–0.69 μmol/mmol cr.
Osmolality350–1000 mosmol/kg
17-Oxogenic steroids ♂28–30 μmol/24h
17-Oxogenic steroids ♀21–66 μmol/24h
17-Oxosteroids ♂17–76 μmol/24h
17-Oxosteroids ♀14–59 μmol/24h
Phosphate (inorganic)15–50 mmol/24h
Potassium14–120 mmol/24h
Protein<150 mg/24h
Protein/creatinine ratio<3 mg/mmol
Sodium100–250 mmol/24h
Haematology
WCC4.0–11.0 ×10⁹/L
RBC ♂4.5–6.5 ×10¹²/L
RBC ♀3.9–5.6 ×10¹²/L
Hb ♂130–180 g/L
Hb ♀115–160 g/L
PCV ♂0.4–0.54 L/L
PCV ♀0.37–0.47 L/L
MCV76–96 fL
MCH27–32 pg
MCHC300–360 g/L
RDW11.6–14.6%
Neutrophils2.0–7.5 ×10⁹/L (40–75%)
Lymphocytes1.0–4.5 ×10⁹/L (20–45%)
Eosinophils0.04–0.44 ×10⁹/L (1–6%)
Basophils0–0.10 ×10⁹/L (0–1%)
Monocytes0.2–0.8 ×10⁹/L (2–10%)
Platelets150–400 ×10⁹/L
Reticulocytes0.8–2.0% / 25–100 ×10⁹/L
Prothrombin time10–14 s
APTT35–45 s
Paediatric
Pulse Rate (bpm)
Neonate140–160
Infant <1yr120–140
1–5 years110–130
5–12 years80–120
>12 years70–100
Respiratory Rate (tachypnoea)
0–2 months≥60/min
2–12 months≥50/min
1–5 years≥40/min
>5 years≥30/min
Blood Pressure (mmHg)
Term65/45
1 year75/50
4 years85/60
8 years95/65
10 years100/70
Weight Formulas
3–12 months(a + 9)/2 kg
1–6 years2a + 8 kg
>6 years(7a − 5)/2 kg
Haemoglobin (g/dL)
Term newborn13–20
1 month11–18
2 months10–15
1–2 years10–13
>2 years11–14
MUAC (6 months–5 years)
Obese>17.5 cm
Normal13.5–17.4 cm
At risk12.5–13.4 cm
Moderate malnutrition11.5–12.4 cm
Severe malnutrition<11.5 cm
Developmental Milestones
Social smile1.5 months
Head control4 months
Sits unsupported7 months
Crawls10 months
Stands unsupported10–12 months
Walks12–13 months
Talks18 months
CSF WBC (/mm³)
Term newborn0–25
>2 weeks0–5
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