Shoulder Dislocation

Last updated: November 17, 2025

Chronic Shoulder Instability and Dislocation

Dislocations make the shoulder joint prone to further episodes. Repeated episodes can lead to chronic shoulder instability where the laxity of the shoulder joint causes the head of the humerus to not remain consistently in its position within the joint.

Stability of the shoulder joint

StabilityDescription
Dynamic stabilityStability during movement. Provided by the rotator cuff
Static stabilityStability at rest. Provided by the glenoid labrum

Anterior Shoulder Dislocation

Anterior dislocation of the head of the humerus from the glenoid cavity is the most common shoulder dislocation (96%). It is frequently seen in younger patients after acute trauma, usually in sports. A history of prior dislocation is a risk factor due to laxity of ligaments.

  • Reasons why anterior shoulder dislocations are so common
    • Shallow glenoid cavity
    • The extraordinary range of movement of the shoulder joint
    • Underlying conditions such as ligamentous laxity
    • The sheer vulnerability of the joint during stressful activity of upper limb
  • Mechanisms
    • Indirect trauma to upper extremity with shoulder in abduction, extension, and external rotation (Most common) aka FOOSH
    • Direct trauma to the posterior shoulder, anteriorly directed
    • Convulsive mechanism and electrical shock
    • Recurrent instability (Congenital or acquired laxity, results in dislocation with minimal trauma)
  • Signs and symptoms
    • Shoulder pain
    • Arm held abducted and externally rotated
    • Prominent head of humerous
    • Notable asymmetry
    • Reduced range of motion at shoulder
  • Physical examination
    • Assess neurovascular status
    • Axillary nerve damage
      • Stabilize the adducted arm and ask the patient to abduct. Palpate the deltoid muscle (motor component)
      • Check for numbness over the regimental patch (skin over the proximal arm; sensory component) ****
    • Axillary artery damage
      • Check for radial pulse
  • Investigations
    • X-ray of affected shoulder: AP view + Scapular-Y (lateral) view, or axillary view
  • Non-operative treatment
    • Closed reduction followed by sling immobilization for 3 weeks
    • NSAIDs for pain and proper healing of the ligaments
  • Techniques for closed reduction
    • Stimson’s technique: the patient is left prone with the arm hanging over the side of the bed. Give high analgesics. After 15 or 20 minutes the shoulder may reduce
    • Hippocratic method: gently increasing traction is applied to the arm with the shoulder in slight abduction, while an assistant applies firm countertraction to the body
    • Kocher’s method: The elbow is bent to 90 degrees and held close to the body; no traction should be applied. The arm is slowly rotated 75 degrees laterally, then the point of the elbow is lifted forwards, and finally the arm is rotated medially. This technique carries the risk of nerve, vessel and bone injury and is not recommended.
  • Indications for operative management
    • First-time dislocation in young active men
    • Soft tissue interposition
    • Labral injury
    • Diplaced greater tuberosity fracture that remains >5mm superiorly displaced following closed reduction
    • Glenoid rim fracture >5mm in size
  • Operative treatment
    • Arthroscopic ligamentous repair of anterior/inferior labrum (Bankart lesion)
    • Shoulder immobilizer post-op for upto 3 weeks
  • Complications of anterior shoulder dislocation
    • Recurrent anterior dislocation
    • Osseous lesions
      • Hill-Sachs lesion (compression fracture of the posterolateral humeral head caused by impaction against the glenoid rim)
      • Glenoid lip fracture (”Bony Bankart Lesion”)
      • Greater tuberosity fracture
      • Fracture of acromion or coracoid
      • Post-traumatic degenerative changes
    • Soft-tissue lesions
      • Rotator cuff tear (older patients)
      • Capsular or subscapularis tendon tear
    • Vascular injury (Axillary artery)
    • Nerve injury (Musculocutaneous and axillary nerves)
    • Late complications
      • Shoulder stiffness
      • Unreduced dislocation
      • Recurrent dislocation: can be predicted through the apprehension test

Associated injuries

InjuryDescription
Bankart lesionA tear or detarchment of the anterior-inferior portion of the glenoid labrum from the glenoid rim (caused by anterior dislocation)
Osseus Bankart lesionA fracture of the anterior-inferior portion of the glenoid bone in association with a Bankart lesion of the labrum
Reverse Bankart lesionA tear of the posterior-inferior portion of the glenoid labrum (caused by posterior dislocation)
Perthes lesionA variant of Bankart lesion where the anterior-inferior laburm is detached but remains attached to the intact peritoneum
Anterior Labroligamentous Periosteal Sleeve Avulsion (ALPSA)A variant of Bankart lesiosn in which the anterior-inferior labrum is torn and medially dislpaced along with the intact periosteum
Glenolabral Articular Disruption (GLAD)A superficial injury involving the anterior-inferior labrum and adjacent glenoid articular cartilage, without significant instability

Posterior Shoulder Dislocation

Posterior dislocation of the head of the humerus from the glenoid cavity is extremely rare (2-4% of shoulder dislocations). It is easy to miss and usually results from disorganised rotator cuff muscle contraction (such as in electric shock, seizures, etc.) or from severe restraint, e.g. half-Nelson

  • Mechanism
    • Indirect trauma with the shoulder in adduction, flexion, and internal rotation
    • Electric shock or convulsive mechanism (Contraction of big internal rotators > External rotators)
    • Direct trauma applied to anterior shoulder
  • Signs and symptoms
    • Painful shoulder
    • Arm held adducted and internally rotated and locked in that position
    • Palpable mass posterior to shoulder
    • Flattened anterior shoulder
    • Coracoid prominence
  • Investigations
    • X-ray of affected shoulder (AP, scapular-Y, and axillary views): often undiagnosed with unidirectional XR
      • Light bulb appearance on AP view, as the head of humerus stands somewhat away from the glenoid cavity (empty glenoid sign)
  • Treatment
    • Non-operative
      • Closed reduction and sling immobilisation
      • Reduction is by pulling on the arm with the shoulder in adduction, then gently rotating laterally while the head is pushed forwards
      • NSAIDs for pain
    • Operative
      • Open reduction
Reference Intervals
Biochemistry
ACTHP: <80 ng/L
ALTP: 5–35 U/L
AlbuminP: 35–50 g/L
AldosteroneP: 100–500 pmol/L
Alk. phosphataseP: 30–130 U/L
α-AmylaseP: 0–180 IU/dL
α-FetoproteinS: <10 kU/L
Angiotensin IIP: 5–35 pmol/L
ADHP: 0.9–4.6 pmol/L
ASTP: 5–35 U/L
BicarbonateP: 24–30 mmol/L
BilirubinP: 3–17 μmol/L
BNPP: <50 ng/L
CRPP: <10 mg/L
CalcitoninP: <0.1 mcg/L
Calcium (ionized)P: 1.0–1.25 mmol/L
Calcium (total)P: 2.12–2.60 mmol/L
ChlorideP: 95–105 mmol/L
CholesterolP: <5.0 mmol/L
VLDLP: 0.128–0.645 mmol/L
LDLP: <2.0 mmol/L
HDLP: 0.9–1.93 mmol/L
Cortisol AMP: 450–700 nmol/L
Cortisol MidnightP: 80–280 nmol/L
CK ♂P: 25–195 U/L
CK ♀P: 25–170 U/L
CreatinineP: 70–100 μmol/L
FerritinP: 12–200 mcg/L
FolateS: 2.1 mcg/L
FSHP: 2–8 U/L ♂; >25 menopause
GGT ♂P: 11–51 U/L
GGT ♀P: 7–33 U/L
Glucose (fasting)P: 3.5–5.5 mmol/L
Growth hormoneP: <20 mu/L
HbA1C (DCCT)B: 4–6%
HbA1C (IFCC)B: 20–42 mmol/mol
Iron ♂S: 14–31 μmol/L
Iron ♀S: 11–30 μmol/L
Lactate (venous)P: 0.6–2.4 mmol/L
Lactate (arterial)P: 0.6–1.8 mmol/L
LDHP: 70–250 U/L
LHP: 3–16 U/L
MagnesiumP: 0.75–1.05 mmol/L
OsmolalityP: 278–305 mosmol/kg
PTHP: 0.8–8.5 pmol/L
PotassiumP: 3.5–5.3 mmol/L
Prolactin ♂P: <450 U/L
Prolactin ♀P: <600 U/L
PSAP: 0–4 mcg/mL
Protein (total)P: 60–80 g/L
Red cell folateB: 0.36–1.44 μmol/L
Renin (erect)P: 2.8–4.5 pmol/mL/h
Renin (recumbent)P: 1.1–2.7 pmol/mL/h
SodiumP: 135–145 mmol/L
TBGP: 7–17 mg/L
TSHP: 0.5–4.2 mU/L
T4P: 70–140 nmol/L
Free T4P: 9–22 pmol/L
TIBCS: 54–75 μmol/L
TriglyceridesP: 0.50–2.3 mmol/L
T3P: 1.2–3.0 nmol/L
Troponin TP: <0.1 mcg/L
Urate ♂P: 210–480 μmol/L
Urate ♀P: 150–390 μmol/L
UreaP: 2.5–6.7 mmol/L
Vitamin B12S: 0.13–0.68 nmol/L
Vitamin DS: 50 nmol/L
Arterial Blood Gases
pH7.35–7.45
PaCO₂4.7–6.0 kPa
PaO₂>10.6 kPa
Base excess±2 mmol/L
Urine
Cortisol (free)<280 nmol/24h
Hydroxyindole acetic acid16–73 μmol/24h
Hydroxymethylmandelic acid16–48 μmol/24h
Metanephrines0.03–0.69 μmol/mmol cr.
Osmolality350–1000 mosmol/kg
17-Oxogenic steroids ♂28–30 μmol/24h
17-Oxogenic steroids ♀21–66 μmol/24h
17-Oxosteroids ♂17–76 μmol/24h
17-Oxosteroids ♀14–59 μmol/24h
Phosphate (inorganic)15–50 mmol/24h
Potassium14–120 mmol/24h
Protein<150 mg/24h
Protein/creatinine ratio<3 mg/mmol
Sodium100–250 mmol/24h
Haematology
WCC4.0–11.0 ×10⁹/L
RBC ♂4.5–6.5 ×10¹²/L
RBC ♀3.9–5.6 ×10¹²/L
Hb ♂130–180 g/L
Hb ♀115–160 g/L
PCV ♂0.4–0.54 L/L
PCV ♀0.37–0.47 L/L
MCV76–96 fL
MCH27–32 pg
MCHC300–360 g/L
RDW11.6–14.6%
Neutrophils2.0–7.5 ×10⁹/L (40–75%)
Lymphocytes1.0–4.5 ×10⁹/L (20–45%)
Eosinophils0.04–0.44 ×10⁹/L (1–6%)
Basophils0–0.10 ×10⁹/L (0–1%)
Monocytes0.2–0.8 ×10⁹/L (2–10%)
Platelets150–400 ×10⁹/L
Reticulocytes0.8–2.0% / 25–100 ×10⁹/L
Prothrombin time10–14 s
APTT35–45 s
Paediatric
Pulse Rate (bpm)
Neonate140–160
Infant <1yr120–140
1–5 years110–130
5–12 years80–120
>12 years70–100
Respiratory Rate (tachypnoea)
0–2 months≥60/min
2–12 months≥50/min
1–5 years≥40/min
>5 years≥30/min
Blood Pressure (mmHg)
Term65/45
1 year75/50
4 years85/60
8 years95/65
10 years100/70
Weight Formulas
3–12 months(a + 9)/2 kg
1–6 years2a + 8 kg
>6 years(7a − 5)/2 kg
Haemoglobin (g/dL)
Term newborn13–20
1 month11–18
2 months10–15
1–2 years10–13
>2 years11–14
MUAC (6 months–5 years)
Obese>17.5 cm
Normal13.5–17.4 cm
At risk12.5–13.4 cm
Moderate malnutrition11.5–12.4 cm
Severe malnutrition<11.5 cm
Developmental Milestones
Social smile1.5 months
Head control4 months
Sits unsupported7 months
Crawls10 months
Stands unsupported10–12 months
Walks12–13 months
Talks18 months
CSF WBC (/mm³)
Term newborn0–25
>2 weeks0–5
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