Rectal Prolapse - Hyperexcision

Last updated: November 17, 2025

Table Of Contents

Rectal Prolapse (Procidentia)
Internal rectal prolapse (”Internal intussusception”)

Rectal Prolapse (Procidentia)

A rectal prolapse is an umbrella term that refers to the abnormal full-thickness protrusion of the rectum through the anus. It can be easy to confuse rectal prolapse with prolapsed haemorrhoids. One way to differentiate a rectal prolapse from prolapsed haemorrhoids is the direction of mucosal folds – mucosal folds are oriented in a circular direction for rectal prolapse, while in haemorrhoids the folds are oriented radially.

Prolapse is common in older women. Important factors such as chronic constipation and childbirth are associated with prolapse.

Types of rectal prolapse.

Prolapse	Description
Mucosal rectal prolapse	Only the mucosal layer prolapses past anal verge. Noted by radial folds (like prolapsed hemorrhoids). Common in children and treatment is usually conservative. Predisposing factors include malnutrition (loss of fat in the ischiorectal fossa), direct downward course of the rectum in infants and diarrhoea.
Full rectal prolapse (procidentia)	All layers of the rectum prolapse past the anal verge. Noted by circumferential folds. Common in adults and is associated with incontinence. May occur in children with malnutrition. This type requires surgery.

Patient history
- Bowel habits (constipation or incontinence – reflects poor anal tone)
Physical examination
- Sphincter tone
Signs and symptoms
- Mass protruding past anal verge (during defecation and valsava).
  - Spontaneously reduces
  - Radial folds (mucosal) or circumferential folds (full)
- Fecal incontinence (28-88%)
Differentials
- Hemorrhoids: appreciated as a mass with radial folds; may be painful (external) or bleed (internal)
Investigations
- Anoscopy or proctosigmoidoscopy; to visualize the rectum. Findings can alter the surgical plan
Treatment of mucosal rectal prolapse
- Treat the underlying cause
- Digital repositioning by the parents
- Submucosal injection (using 6% phenol in almon oil) or rubber band ligation if digital repositioning fails
Treatment of full rectal prolapse (procidentia)
- Altemeier procedure (perineal approach): involves resecting the rectum through the perineum and tightening the anal sphincter. Procedure of choice for incarcerated and strangulated prolapse. Complications include fecal and flatus incontinence due to loss of rectal reservoir.
- Delomre’s procedure (perineal approach): the mucosa is stripped circumferentially then anastomosed to reduce the prolapse. This has a high recurrence rate
- Rectopexy(abdominal approach): fix the redundant rectosigmoid in the pelvis by sutures or slings or mesh or fascia) e.g. Ripstein procedure
- Resection of the redundant sigmoid

Internal rectal prolapse (”Internal intussusception”)

An internal rectal prolapse is the telescoping of the rectum distally, usually not protruding past the anal verge. It is common in paediatric patients.

Things to consider in paediatric patients with rectal prolapse
- Whipworm infection
- Clostridiodes difficile infection
- Cystic fibrosis in white kids
Signs and symptoms
- Urgency to defecate
- Feeling of rectal fullness
- “Constipation”
- Pelvic pain
Investigations
- High-resolution ultrasound
- Pelvic MRI
- Proctosigmodioscopy: always done, to rule out other anorectal pathologies
- Additional investigations: stool ova and cysts, c-difficile toxin, sweat chloride studies
Treatment
- Treat the underlying cause
- Stool softeners
- Increase dietary fibre intake
- Surgery is rarely indicated

Reference Intervals ›

Biochemistry

ACTH	P: <80 ng/L
ALT	P: 5–35 U/L
Albumin	P: 35–50 g/L
Aldosterone	P: 100–500 pmol/L
Alk. phosphatase	P: 30–130 U/L
α-Amylase	P: 0–180 IU/dL
α-Fetoprotein	S: <10 kU/L
Angiotensin II	P: 5–35 pmol/L
ADH	P: 0.9–4.6 pmol/L
AST	P: 5–35 U/L
Bicarbonate	P: 24–30 mmol/L
Bilirubin	P: 3–17 μmol/L
BNP	P: <50 ng/L
CRP	P: <10 mg/L
Calcitonin	P: <0.1 mcg/L
Calcium (ionized)	P: 1.0–1.25 mmol/L
Calcium (total)	P: 2.12–2.60 mmol/L
Chloride	P: 95–105 mmol/L
Cholesterol	P: <5.0 mmol/L
VLDL	P: 0.128–0.645 mmol/L
LDL	P: <2.0 mmol/L
HDL	P: 0.9–1.93 mmol/L
Cortisol AM	P: 450–700 nmol/L
Cortisol Midnight	P: 80–280 nmol/L
CK ♂	P: 25–195 U/L
CK ♀	P: 25–170 U/L
Creatinine	P: 70–100 μmol/L
Ferritin	P: 12–200 mcg/L
Folate	S: 2.1 mcg/L
FSH	P: 2–8 U/L ♂; >25 menopause
GGT ♂	P: 11–51 U/L
GGT ♀	P: 7–33 U/L
Glucose (fasting)	P: 3.5–5.5 mmol/L
Growth hormone	P: <20 mu/L
HbA1C (DCCT)	B: 4–6%
HbA1C (IFCC)	B: 20–42 mmol/mol
Iron ♂	S: 14–31 μmol/L
Iron ♀	S: 11–30 μmol/L
Lactate (venous)	P: 0.6–2.4 mmol/L
Lactate (arterial)	P: 0.6–1.8 mmol/L
LDH	P: 70–250 U/L
LH	P: 3–16 U/L
Magnesium	P: 0.75–1.05 mmol/L
Osmolality	P: 278–305 mosmol/kg
PTH	P: 0.8–8.5 pmol/L
Potassium	P: 3.5–5.3 mmol/L
Prolactin ♂	P: <450 U/L
Prolactin ♀	P: <600 U/L
PSA	P: 0–4 mcg/mL
Protein (total)	P: 60–80 g/L
Red cell folate	B: 0.36–1.44 μmol/L
Renin (erect)	P: 2.8–4.5 pmol/mL/h
Renin (recumbent)	P: 1.1–2.7 pmol/mL/h
Sodium	P: 135–145 mmol/L
TBG	P: 7–17 mg/L
TSH	P: 0.5–4.2 mU/L
T4	P: 70–140 nmol/L
Free T4	P: 9–22 pmol/L
TIBC	S: 54–75 μmol/L
Triglycerides	P: 0.50–2.3 mmol/L
T3	P: 1.2–3.0 nmol/L
Troponin T	P: <0.1 mcg/L
Urate ♂	P: 210–480 μmol/L
Urate ♀	P: 150–390 μmol/L
Urea	P: 2.5–6.7 mmol/L
Vitamin B12	S: 0.13–0.68 nmol/L
Vitamin D	S: 50 nmol/L

Arterial Blood Gases

pH	7.35–7.45
PaCO₂	4.7–6.0 kPa
PaO₂	>10.6 kPa
Base excess	±2 mmol/L

Urine

Cortisol (free)	<280 nmol/24h
Hydroxyindole acetic acid	16–73 μmol/24h
Hydroxymethylmandelic acid	16–48 μmol/24h
Metanephrines	0.03–0.69 μmol/mmol cr.
Osmolality	350–1000 mosmol/kg
17-Oxogenic steroids ♂	28–30 μmol/24h
17-Oxogenic steroids ♀	21–66 μmol/24h
17-Oxosteroids ♂	17–76 μmol/24h
17-Oxosteroids ♀	14–59 μmol/24h
Phosphate (inorganic)	15–50 mmol/24h
Potassium	14–120 mmol/24h
Protein	<150 mg/24h
Protein/creatinine ratio	<3 mg/mmol
Sodium	100–250 mmol/24h

Haematology

WCC	4.0–11.0 ×10⁹/L
RBC ♂	4.5–6.5 ×10¹²/L
RBC ♀	3.9–5.6 ×10¹²/L
Hb ♂	130–180 g/L
Hb ♀	115–160 g/L
PCV ♂	0.4–0.54 L/L
PCV ♀	0.37–0.47 L/L
MCV	76–96 fL
MCH	27–32 pg
MCHC	300–360 g/L
RDW	11.6–14.6%
Neutrophils	2.0–7.5 ×10⁹/L (40–75%)
Lymphocytes	1.0–4.5 ×10⁹/L (20–45%)
Eosinophils	0.04–0.44 ×10⁹/L (1–6%)
Basophils	0–0.10 ×10⁹/L (0–1%)
Monocytes	0.2–0.8 ×10⁹/L (2–10%)
Platelets	150–400 ×10⁹/L
Reticulocytes	0.8–2.0% / 25–100 ×10⁹/L
Prothrombin time	10–14 s
APTT	35–45 s

Paediatric

Pulse Rate (bpm)
Neonate	140–160
Infant <1yr	120–140
1–5 years	110–130
5–12 years	80–120
>12 years	70–100
Respiratory Rate (tachypnoea)
0–2 months	≥60/min
2–12 months	≥50/min
1–5 years	≥40/min
>5 years	≥30/min
Blood Pressure (mmHg)
Term	65/45
1 year	75/50
4 years	85/60
8 years	95/65
10 years	100/70
Weight Formulas
3–12 months	(a + 9)/2 kg
1–6 years	2a + 8 kg
>6 years	(7a − 5)/2 kg
Haemoglobin (g/dL)
Term newborn	13–20
1 month	11–18
2 months	10–15
1–2 years	10–13
>2 years	11–14
MUAC (6 months–5 years)
Obese	>17.5 cm
Normal	13.5–17.4 cm
At risk	12.5–13.4 cm
Moderate malnutrition	11.5–12.4 cm
Severe malnutrition	<11.5 cm
Developmental Milestones
Social smile	1.5 months
Head control	4 months
Sits unsupported	7 months
Crawls	10 months
Stands unsupported	10–12 months
Walks	12–13 months
Talks	18 months
CSF WBC (/mm³)
Term newborn	0–25
>2 weeks	0–5

Calculator ›

Rectal Prolapse (Procidentia)

Internal rectal prolapse (”Internal intussusception”)

Post DiscussionCancel Reply