Cerebral Palsy

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Cerebral Palsy

Cerebral palsy is a neurological disorder caused by non-progressive brain injury or malformation resulting in permanent neurological damage. This occurs when the child’s brain is developing. Body movement and muscle coordination is primarily affected. The most common cause is anoxic brain injury during the perinatal perior or before 2 years of age. The aim of treating cerebral palsy is to support the patient to achieve the most fulfilled and independent life. Management involves a multi-disciplinary team approach

Causes of cerebral palsy

CauseExamples
AntenatalMaternal infection, trauma during pregnancy
PerinatalBirth asphyxia, pre-term birth
Post-natalMeningitis, intraventricular hemorrhage, severe neonatal jaundice, head injury

Classification of cerebral palsy

ClassificationDescription
Spastic CP (Pyramidal, 75%)Increased muscle tone with increased muscle velocity (hypertonia) and hyperreflexia. Most common subtype. Due to damage of upper motor neurons.
Athetoid (Dyskinetic/Extrapyramidal)Problems controlling muscle tone with hypertonia and hypotonia which causes Involunatry writhing movements and oro-motor problems. Results from damage to the basal ganglia.
AtaxicIssues with coordination and balance. Results from damage to the cerebellum
HypotonicLoss of muscle tone and strength, resulting in weakness and floppiness
MixedMix of spastic, dyskinetic and/or ataxic features due to damage to different portions of the brain

Classification according to limbs affected

ClassificationDescription
QuadriplegicAll 4 extremities are affected severely. Also often involves muscles of facial expression, seizures, speech disturbances and other impairments.
DiplegicTwo contralateral extremitites (typically both lower extremitites) are affected
HemiplegicTwo ipsilateral extremities are affected
MonoplegicOne limb is affected
  • Signs and symptoms
    • Failure to meet milestones
    • Increased or decreased tone (generally or in affected limbs)
    • Hand preference before 18 months
    • Problems with coordination, speech or walking
    • Feeding or swallowing problems
    • Learning difficulties
  • Physical examination
    • Hemiplegic/diplegic gait (UMN lesion): legs extended with plantarflexion of the feet and toes. The leg is swung around in a large semicircle when it is moved from behind to front.
    • Good muscle bulk
    • Hypertonia
    • Spasticity (muscle stiffness or tightness that increases with muscle contraction velocity)
    • Brisk reflexes
    • Slightly reduced power
    • Athetoid movements
    • Test for coordination
  • Non-operative treatment
    • Physiotherapy: to stretch and strengthen muscles, maximize function and prevent contractures
    • Occupational therapy: to help the patients manage everyday activities
    • Speech and language therapy: to help with speech and swallowing
    • Dieticians: to ensure they meet nutritional requirements. Some children can be fed using a PEG or NG tube
    • Mobility assistance e.g. walkers
    • Bracing
    • Social worker: to help with benefits and support
    • Charities and support groups to provide opportun
  • Pharmacological treatment
    • Muscle relaxants (baclofen) for spasticity and contracutres
    • Anti-epileptic drugs for seizures
    • Glycopyrrolate for excessive drooling
  • Indications and options for operative treatment (orthopaedics)
    • Hip sublaxation or dislocation: tendon release and osteotomies
    • Knee contractures: hamstring lengthening procedures
    • Foot and ankle deformitites: tendon transfers or osteotomies
  • Complications and associated conditions
    • Learning disability
    • Epilepsy
    • Kyphoscoliosis
    • Muscle contractures
    • Hearing and visual impairment
    • Gastro-esophageal reflux

Examining for Spasticity

Jeffrey Kalei
Jeffrey Kalei
Articles: 335

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